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. 2022 Aug 30;17:102. doi: 10.1186/s13020-022-00648-x

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — FTZ protects HK-2 cells by activating Nrf2 pathway and inhibiting apoptosis a The expression of Nrf2, HO-1, and NQO1 protein in the HK-2 cells. b The expression of Bax and Bcl-2 protein in the HK-2 cells. Model group: the HK-2 cells were exposed to 200 µM H₂O_2; FTZ group: the HK-2 cells were exposed to 200 µM H₂O₂ for 24 h after 150 µg/mL FTZ treatment for 6 h. Values are the mean ± SEM. ^#P < 0.05, ^##P < 0.01(compared to Ctrl group); ^*P < 0.05, ^**P < 0.01 (compared to Mod group) (n = 3)