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. 2022 Aug 19;13:970269. doi: 10.3389/fendo.2022.970269

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Copyright © 2022 Li, Luo, Wang, Zeng, Feng and Che

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Identification of radiotherapy-associated CRGs. (A) Box plot showing Cuproptosis pathway activity before and after radiotherapy. (B) Identification of the most relevant gene modules for Cuproptosis. (C) Heat map showing genomic changes and hazard ratios of CRGs in TCGA-LUAD. From left to right were respectively: expression differences of CRGs in tumor and normal samples, frequency of mutations and copy number variants of CRGs, correlation of DNA methylation modifications and expression of CRGs, and univariate Cox regression analysis showing risk ratios of CRGs. *p < 0.05, **p < 0.01, ***p < 0.001. (D) Summary of mutational events in CRGs in TCGA-LUAD. (E) Circle diagram demonstrating the CNV events of CRGs on chromosomes.