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. 2022 Sep 8;41:270. doi: 10.1186/s13046-022-02478-z

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — SiLINC01132 improves the response to anti-PDL1 immunotherapy in a subgroup of HCC. A Scatter plots showing the correlation between LINC01132 expression and CD8+ T cells infiltrations. B Mice were orthotopically xenografted with Hep1–6 injection and treated with anti-PDL1 or IgG or siLINC01132 plus anti-PDL1. Bottom panel showing representative images of tumors treated with IgG + shLINC01132 or anti-PDL1 + shLINC01132. C-D The tumor volume and weight of mice treated with IgG + shLINC01132 or anti-PDL1 + shLINC01132. C for tumor volume and D for tumor weight. E IHC staining of CD8A and the number of CD8A positive cells. F The mechanistic scheme of lncRNA LINC01132/NRF1/DPP4 axis in HCC