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. 2022 Aug 26;13:992743. doi: 10.3389/fimmu.2022.992743

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Copyright © 2022 Flores-Lovon, Ortiz-Saavedra, Cueva-Chicaña, Aperrigue-Lira, Montes-Madariaga, Soriano-Moreno, Bell and Macedo

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The immune response in COVID-19 and active tuberculosis. The Th1 response potentiates macrophage activity and the Th17 response favors the recruitment and activation of neutrophils with release of harmful products to the host tissue, further exacerbating the inflammatory process. To appease these responses, an elevated production of IL-10 and differentiation of Treg occurs, however, this generates a low immunologic potential. This response caused by M. tuberculosis generates an unfavorable immune microenvironment for theresnponse against SARS-CoV-2. Created with BioRender.com.