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. 2022 Sep 8;13:465. doi: 10.1186/s13287-022-03157-8

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — The IFN-γ and KYNA improved hADSCs homing to the colon tissue. (A) The morphology of Dil-labeled hADSCs and primed hADSCs was observed under microscope (scale bar, respectively, 50, 100, 10 μm); (B) The rats were killed on day 10 and the fluorescence signals of Dil-labeled cells in the colon and small intestine were observed under the IVIS system; (C) In vivo distribution of hADSCs and primed hADSCs was observed by immunofluorescence staining of the colon, lung, liver, and spleen. The white arrow points to the Dil-labeled hADSCs (Scale bar, 50 μm)