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. 2022 Aug;10(16):890. doi: 10.21037/atm-22-3789

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2022 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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Effect of the inhibitor compound C on the endogenous protective mechanism of the body. (A) Myocardial infarct size; (B) CK-MB; (C) LDH; (D) GSH; (E) SOD; (F) ROS. ^a, P<0.05, compared with the Sham group; ^b, P<0.05. compared with the MI/R group. GSH, glutathione; SOD, superoxide dismutase; ROS, reactive oxygen species; MI/R, myocardial ischemia/reperfusion; C, compound C; CK-MB, creatine kinase-MB; LDH, lactate dehydrogenase; INF, myocardial infarct size; AAR, percentage of total ischemia-reperfusion risk area.