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. 2022 Sep 15;20:138. doi: 10.1186/s12958-022-00998-z

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — Circ-CSNK1G1 suppressed the expression of miR-28-5p. A The binding sites between circ-CSNK1G1 and miR-28-5p. B and C Luciferase reporter assay was performed to confirm the relationship between circ-CSNK1G1 and miR-28-5p. D The expression of miR-28-5p in tumor tissues and normal tissues was detected by qPCR. E The expression of miR-28-5p in MCF-10 A, MDA-MB-231 and BT-549 cells was detected by qPCR. F The correlation between circ-CSNK1G1 and miR-28-5p expression was analyzed by Pearson analysis. G The effects of circ-CSNK1G1 silence and overexpression on miR-28-5p expression were detected by qPCR. ***P < 0.001 and ****P < 0.0001