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. 2022 Sep 16;41:275. doi: 10.1186/s13046-022-02475-2

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — ETV1 expression correlates with PTK2 and c-MET expression in HCC specimens. (A) Representative IHC staining of ETV1, PTK2, and c-MET in primary HCC specimens and paired nontumorous specimens. (B) Association between ETV1 and PTK2 or c-MET expression in two HCC cohorts. (C-D) Kaplan-Meier curves showed the correlations between PTK2, c-MET, ETV1/PTK2, or ETV1/c-MET expression and OS or recurrence rates. * Represented p < 0.05. All data were displayed as Mean ± SD