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. 2022 Sep 17;23:249. doi: 10.1186/s12931-022-02172-w

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — a In lung tissues, LPO levels were significantly increased in the OA group (P < 0.05) and were significantly decreased in the DEX and TCZ groups (P < 0.05). b SOD activity was significantly decreased in the OA and OA + TCZ-2 groups (P < 0.05), while it was significantly increased in the OA + TCZ-4 and OA + DEX-10 groups compared to the OA group (P < 0.05). c In the OA + TCZ-4 group, GSH levels were significantly increased compared to the OA and other experimental groups (P < 0.05). d CAT activity was significantly decreased in the OA group (P < 0.05), while it was significantly increased in all other experimental groups compared to the OA group (P < 0.05). Different letters (a, b, c, d) above bars indicate statistical differences between control and experimental groups according to Tukey’s multiple range test (P < 0.05)