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. 2022 Sep 19;22:287. doi: 10.1186/s12935-022-02712-w

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Knockdown of YY1 impairs cellular nucleotide homeostasis. A Metabolite profiling results corresponding to MIA-PaCa2 control and YY1 KD cells. The red and blue points represent up- and down-regulated YY1 KD cell metabolites, respectively (P < 0.05, n = 6 per group). B Metabolite Sets Enrichment analysis of whole metabolites. C Pathway impact analysis of whole metabolites, pathways with P < 0.05 were marked in the figure. D DNA content in MIA-PaCa2 control and YY1 KD cells (n = 3 per group). E Statistical analysis of the cell cycle