Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2022 Sep 12;2022:2852251. doi: 10.1155/2022/2852251

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

Copyright © 2022 Guoxia Zhang et al.

This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

PMC Copyright notice

The mechanisms of EXOs against DIC involved in anti-inflammation, and antipyroptosis. MSC: mesenchymal stem cell; MSC-EXO: exosome derived from mesenchymal stem cell; CPC: cardiac progenitor cell; CPC-EXO: exosome derived from cardiac progenitor cell; EC: endothelial cell; EC-EV: extracellular vesicle derived from endothelial cell; JAK2: janus kinase 2; STAT6: signal transducer and activator of transcription 6; Rac1: ras-related C3 botulinum toxin substrate 1; TNF-α: tumor necrosis factor-alpha; IL-6: interleukin-6; IL-1β: interleukin-1β; ESC: embryonic stem cell; ESC-EXO: exosome derived from embryonic stem cell; TLR4: toll-like receptor-4; NLRP3: NLR family pyrin domain containing 3; KLF2: Krüppel-like factor 2.