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. 2022 Sep 20;13(9):806. doi: 10.1038/s41419-022-05243-4

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a Crossing of Snf5‐related 1 (Snr1) knockdown flies with strains expressing specific RNA interference (RNAi) shifted the pupal lethal phenotype associated with glial‐specific Snr1 knockdown to later stages of development in 14 out of 59 screened candidate genes. The complete list of the 59 genes is shown in Supplementary Table 8. Created with BioRender.com. b Kaplan‐Meier survival curve of NOD scid gamma (NSG) mice orthotopically injected with control BT‐12 cells (n = 6, shCTRL, blue) or KIF3A knockdown BT‐12 cells (n = 6, shKIF3A, red). Statistical analysis was performed using the Gehan‐Breslow‐Wilcoxon test.