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. 2022 Aug 29;11(9):1697. doi: 10.3390/antiox11091697

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Protective role of OLNZ on gene expression of complement components in the hippocampus during transient-ischemia (TI)-induced stroke in gerbils. In TI-induced stroke, gene expression of C1q, C2, C3, C4a, and C9, was significantly upregulated, whereas treatment with OLNZ markedly downregulated the expression of these genes. Data were expressed as the mean ± SEM, n = 3 in each group. After performing a one-way ANOVA with Tukey’s post hoc test, p < 0.05 for comparisons of treatment groups with the sham group (#) and p < 0.05 for comparisons with the stroke group (*) were considered significant.