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. 2022 Jun 8;45(5):2078–2090. doi: 10.1007/s10753-022-01677-2

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — Dapagliflozin alleviates myocarditis through activating stat3 signal pathway. a Western blot detection of iNOS, IL-6, pstat3, stat3, and GAPDH expression. Statistics of b pstat3/stat3, c iNOS, d IL-6. e Western blot detection of iNOS, IL-6, pstat3, stat3, and GAPDH expression. Statistics of f pstat3/stat3, g iNOS, h IL-6. Data were expressed as mean ± SD. All the experiments were repeated at least three times (control, normal mice; Dapa, normal mice treated with dapagliflozin; CVB3, CVB3-infected mice treated with 60% propylene glycol; CVB3 + Dapa, CVB3-infected mice treated with dapagliflozin. CVB3 + Dapa + STATTIC, CVB3-infected mice treated with dapagliflozin and STATTIC. *P < 0.05, **P < 0.01, ***P < 0.001, ****P < 0.0001).