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. 2022 May 31;30(5):1745–1758. doi: 10.1007/s10787-022-01004-z

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 10 — Effect of diapocynin administration on 3-NP-induced immunohistochemical expression of GFAP. Photomicrographs of control group (A, E), diapocynin group (B, F), 3-NP group (C, G), and 3-NP + diapocynin group (D, H). Black arrows indicate GFAP immunoexpression. Magnifications: ×100 (A–D) and ×400 (E–H). I A bar chart representing the mean percentage of GFAP immunoexpression in each group with each bar with vertical line illustrating the mean ± S.E.M. of 3 rats per group. Data were analyzed using one-way ANOVA followed by Tukey’s post hoc test. ^*p < 0.05 vs. control, ^@p < 0.05 vs. 3NP group. 3-NP 3-nitropropionic acid, GFAP glial fibrillary acidic protein