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. Author manuscript; available in PMC: 2022 Sep 23.
Published in final edited form as: Hepatol Int. 2016 May 18;10(6):937–946. doi: 10.1007/s12072-016-9733-6

Fig. 2.

Fig. 2

a, b IFN signaling and JAK-STAT pathway such as STAT1, STAT2 and IRF7 SOCS3, OAS2, and USP18 (negative regulator), were highly expressed in HCV GT-3 versus NASH. c Other important ISGs such as IFI27, IFI44, Mx1 and ISG15 were significantly increased in HCV GT-3 compared to NASH. Values are mean ± SE of the delta Ct values. p value was calculated by the unpaired t test with Welch’s correction using GraphPad Prism 6.0 software. p values less than 0.05 were considered significant