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. 2022 Aug 30;19(10):1130–1140. doi: 10.1038/s41423-022-00904-y

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Ruxolitinib reduced the expression of proinflammatory cytokine genes in the livers of ARE-Del^+/− mice. RNA was isolated from the livers of ruxolitinib-treated mice (n = 11) and control mice (n = 8). The levels of mRNA expression of the proinflammatory genes (IFNγ, IL-6, and TNFα) and, for purposes of comparison, TGFβ were quantified by real-time PCR. The data displayed are expressed as the mean ± SEM. *p < 0.05, ***p < 0.001