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Increased mitochondrial but not nuclear TERT restores physiological NO in vessels from patients with CAD. (A) In vessels from patients with CAD, overexpression of the MTS mutant TERT has no affect on the mechanism of vasodilation. (B) Overexpression of the NLS mutant TERT changed the mechanism of FMD from H2O2 (Peg-catalase inhibitable) to NO (L-NAME inhibitable). (C) Smooth muscle dependent dilation to papaverine was not impaired. * P < 0.05 2-way ANOVA RM, N = 6–8.
