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. 2022 Sep 20;11:e74777. doi: 10.7554/eLife.74777

Table 1. Clinical characteristics and genetic ancestry of the discovery cohort.

PUV (n=132) Controls (n=23,727)
Median age (range) 13 (2–66)
Males (%) 132 (100) 10,425 (43.9)
PCA determined ancestry
EUR (%) 89 (67.4) 19,418 (81.8)
SAS (%) 18 (13.6) 2847 (12.0)
AFR (%) 11 (8.3) 449 (1.9)
AMR (%) 0 (0) 7 (0.03)
Admixed (%) 14 (10.6) 1006 (4.2)
Additional renal/urinary phenotypes
Hydronephrosis (%) 56 (42.4)
Bladder abnormality (%) 32 (24.2)
Hydroureter (%) 30 (22.7)
VUR (%) 27 (20.5)
Renal dysplasia (%) 16 (12.1)
Hypertension (%) 11 (8.3)
Renal agenesis (%) 8 (6.1)
Recurrent UTIs (%) 5 (3.8)
Renal hypoplasia (%) 4 (3.0)
Renal duplication (%) 2 (1.5)
Extrarenal manifestations (%) 35 (26.5)
Cardiac anomaly (%) 4 (3.0)
Neurodevelopmental disorder (%) 7 (5.3)
Family history (%) 5 (3.8)
End-stage renal disease (%) 23 (17.4)
Median age ESRD (range) 14 (0–39)

PUV, posterior urethral valves; PCA, principal component analysis; EUR, European; SAS, South Asian; AFR, African; AMR, Latino/Admixed American; VUR, vesico-ureteral reflux; UTI, urinary tract infection; ESRD, end-stage renal disease.