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. 2022 Aug 12;35(6):1650–1670. doi: 10.1007/s13577-022-00756-8

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — PINK1 enhances BMSC-mediated repair of kidney tissues in IRI-AKI mice. a Representative image of HE staining, × 200, scale: 100 μm. b Pathological score of renal tubular injury (n = 6). c The levels of BUN in blood samples (n = 6). d The levels of SCR in blood samples (n = 6). e BMSCs were successfully transfected with GFP-PINK1, × 100, BAR: 100 μm. f RFP-BMSCs in kidney tissues were observed with a fluorescence microscope. × 200, BAR: 100 μm. g Quantitative analysis of RFP-BMSCs in injured tissues. SEM, ###p < 0.001, ##p < 0.01 and #p < 0.05, compared with the IRI group; ***p < 0.001, **p < 0.01 and *p < 0.05, compared with among the groups