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. 2022 Aug 12;35(6):1650–1670. doi: 10.1007/s13577-022-00756-8

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — PINK1 enhances the BMSC-mediated reduction in RTEC apoptosis under hypoxia-reoxygenation conditions. a Representative image of apoptosis was examined by FCM. b Quantitative analysis of the 24-h apoptosis rate. c Quantitative analysis of the 48-h apoptosis rate. d Quantitative analysis of the 72-h apoptosis rate. SEM, ###p < 0.001, ##p < 0.01 and #p < 0.05, compared with the IRI group; ***p < 0.001, **p < 0.01 and *p < 0.05, compared with among the groups

Fig. 5 — PINK1 enhances the BMSC-mediated reduction in RTEC apoptosis under hypoxia-reoxygenation conditions. a Representative image of apoptosis was examined by FCM. b Quantitative analysis of the 24-h apoptosis rate. c Quantitative analysis of the 48-h apoptosis rate. d Quantitative analysis of the 72-h apoptosis rate. SEM, ###p < 0.001, ##p < 0.01 and #p < 0.05, compared with the IRI group; ***p < 0.001, **p < 0.01 and *p < 0.05, compared with among the groups