Fig. 5.

Model of seedlessness proposed by Royo et al. (Royo et al. 2018) to explain the seedless phenotype in grape. The panel on the left shows the initiation of seed morphogenesis that occurs under normal development. The wild-type VviAGL11 (AGAMOUS-LIKE11) protein complex (shown in purple) either directly or indirectly initiates the expression of genes involved in seed coat sclerification, permitting embryo development. The panel on the right shows a heterozygous individual with a dominant phenotype due to a single missense mutation in the VviAGL11 protein (Arg197 to Leu197; shown in yellow). This mutation prevents the assembly of the multiprotein complex halting seed coat differentiation, leading to degeneration of the embryo and endosperm