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. 2022 Sep 29;39(12):213. doi: 10.1007/s12032-022-01814-9

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Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — VA has been predicted to impact HDAC activity and prostate cancer. “Histone deacetylase activity (H3-K14 specific)” and “histone deacetylase activity” ranked at the first (P < 0.05) and 11th place (P > 0.05), respectively. Both of them have been predicted to be regulated by VA (A). The KEGG pathways analysis (B) displayed the potential diseases or pathways that might be affected by VA. The top predicted result is prostate cancer