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. 2022 Sep 30;20:433. doi: 10.1186/s12967-022-03646-7

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — Transcriptional landscape of TME in the MWA alone and MWA combined with LAG3 blockade groups. A and B. UMAP analysis of all the cells in TME of the MWA alone or MWA combined with the LAG3 blockade group and the proportions of different subpopulations in each sample (MWA group: 8971 cells; MWA combined with the LAG3 blockade group: 8733 cells). C and D. UMAP analysis of all the myeloid cells in TME of the MWA alone or MWA combined with LAG3 blockade group and the proportions of different subpopulations in each sample. E and F. UMAP analysis of all the lymphocytes in TME of the MWA alone or MWA combined with LAG3 blockade group and the proportions of different subpopulations in each sample