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. 2022 Oct 3;13:98. doi: 10.1007/s12672-022-00564-4

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — TGF-β1 regulates morphology migration, invasion and EMT of A549 and NCI-H1299 cells. A Micrographs of A549 and NCI-H1299 cell morphology after their culture in 10 ng/ml TGF-β1 for 48 h (Magnification, × 100). B The expression of E-cadherin and vimentin was evaluated by Western Blot analysis. C Wound healing assay (Magnification, × 100) and D Transwell assay (Magnification, × 200) were performed to assess the cell migration and invasion abilities. ** P < 0.01 compared to control group. Data were expressed as mean ± standard deviation. One-way ANOVA were used to assess differences between groups. The SPSS 25.0 software (USA) used for creation of the figure