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. 2022 Oct 3;13(10):845. doi: 10.1038/s41419-022-05291-w

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — A Oil red O staining indicating the content of LD in OVCAR3-CON, OVCAR3-DHRS2, HO-8910-CON, HO-8910-DHRS2, SKOV3-shCON, and SKOV3-shDHRS2 cells. The mRNA levels of PLA2, PLCL2, PLD1/2, CHKα, and GDPD5/6 in B OVCAR3-CON, OVCAR3-DHRS2 and C SKOV3-shCON and SKOV3-shDHRS2 cells detected by RT-qPCR. D The protein expression levels of CHKα and perilipin1 in OVCAR3-CON, OVCAR3-DHRS2, HO-8910-CON, HO-8910-DHRS2, SKOV3-shCON, and SKOV3-shDHRS2 cells. β-actin was used as a loading control. E The cellular choline content and PC/GPC ratio in OVCAR3-CON and OVCAR3-DHRS2 cells. Data are shown as mean values ± S.D. of independent, triplicate experiments. The asterisks (*,**,***) indicate significant differences (p < 0.05, p < 0.01, p < 0.001, respectively).