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. 2022 Sep 21;10:945586. doi: 10.3389/fcell.2022.945586

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Copyright © 2022 Candido, Tomasello, Lavoro, Falzone, Gattuso, Russo, Paratore, McCubrey and Libra.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Expression and correlation analyses of gene (ENSG) and splicing isoforms (ENST) of LCN2, SLC22A17, and MMP9 in normal and tumoral tissues. (A) Percentage of tumor types in which LCN2, SLC22A17, and MMP9 expression levels were up (red) or downregulated (blue) compared to normal tissues. Only tumors showing FC ≥ 1.4 or ≤ −1.4 (p ≤ 0.05) are included in the analysis. (B) Correlation analysis between LCN2, SLC22A17, and MMP9 genes and isoforms expression in normal and tumor samples. Tumor and normal samples were analyzed collectively. Correlation with Pearson’s r ≥ 0.3 or ≤ −0.3 and p ≤ 0.05 were included in the heatmap. Black bold labels indicate the LCN2, SLC22A17, and MMP9 genes (ENSG). Coding ENST isoforms: blue; non-coding ENST isoforms: green; retained intron ENST isoforms: red.