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. 2022 Oct 7;8:104. doi: 10.1038/s41421-022-00463-6

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a Binding of GW01, 16L9, and 4L12 to the SARS-CoV-2 RBD, Omicron RBD and trimer in an ELISA. REGN10987 was used as a control. b Neutralizing activities of GW01, 16L9, and 4L12 and their combinations against pseudotyped SARS-CoV-2 and its variants Alpha, Beta, Gamma, Delta, and Omicron, as well as sarbecoviruses. S309 was used as a positive control. c Binding of 4L12, 16L9, and RGN10987 to the SARS-CoV-2 RBD in competition with GW01, as measured by BLI.