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. Author manuscript; available in PMC: 2023 Nov 17.
Published in final edited form as: Platelets. 2022 Jun 5;33(8):1119–1131. doi: 10.1080/09537104.2022.2078488

Table 1.

Traumatically-injured patients with increased ApoA-I plasma concentrations.

Ethnicity& Age (years) NISS ACT (sec) Angle (°) MA (mm) Ly30 (%) Mechanism Hosp/ICU/Vent Days Outcome
W 28.3 38 238 49.4 47.5 41.6 Blunt Auto-Ped 1/1/1 Died of TBI
WH 48.9 27 136 70.2 59.4 7.8 Blunt MVC occupant 23/14/11 Pneumonia Discharged
W 28.3 50 160 72.7 56 11.6 Blunt Auto-Ped 18/8/7 α-hemolytic strep Pneumonia Discharged
NA 24.5 27 136 71.1 54.5 3.6 Penetrating GSW 7/4/3 Discharged
Controls 21–54 (n=121) 117 (113–121) 71.4 (68.0–74.2) 61.5 (58.2–64.8) 2.6 (1.6–3.5)

W = White, H = Hispanic, NA = Native American, NISS = New Injury Severity Score, ACT = activated clotting time, MA = maximal amplitude, Ly30 = lysis time 30 after achieving MA, auto = automobile, Ped = pedestrian, MVC = motor vehicle crash, GSW = gunshot wound, Hosp = Hospital days, ICU = Intensive Care Unit days, Vent = ventilator days, TBI = traumatic brain injury. Controls are healthy, male volunteers on no medications that would inhibit platelet function.