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. 2022 Oct 8;20:461. doi: 10.1186/s12967-022-03659-2

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — The expression of PXDN was down-regulated in AKG-treated mice. A Volcano plot showing different gene levels in aortic transcriptome. B Heat map showing levels of genes that encoded proteins having typical oxidoreductase activity along with inflammatory response. C RT-qPCR assay was conducted to analyze PXDN level (n = 4). D Gene ontology (GO) analysis. E KEGG pathway analysis. F ICH was conducted to analyze PXDN level (n = 4). G WB assay and quantitative analysis of PXDN level (n = 4)