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. 2022 Aug 19;127(8):1461–1472. doi: 10.1038/s41416-022-01927-y

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a IHC of ESE3 protein expression in PSCs and adjacent normal pancreatic tissues. b IHC and Masson’s trichrome staining in consecutive PDAC tissue sections. c Double immunohistochemical staining and correlation analysis between ESE3 protein expression and extent of stromal fibrosis (α-SMA, stromal marker) in PDAC samples. d Multiplex IF staining of ESE3 (red), α-SMA (green) and cytokeratin (CK-19, an epithelial marker, purple) in PDAC samples. e IF staining of ESE3 and α-SMA expression in hpPSCs and ihPSCs. f Association of ESE3 expression in PSCs with DFS (left) and OS (right) in PDAC.