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. 2022 Sep 26;146(15):1123–1134. doi: 10.1161/CIRCULATIONAHA.121.058457

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© 2022 The Authors.

Circulation is published on behalf of the American Heart Association, Inc., by Wolters Kluwer Health, Inc. This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited.

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Figure 2. — Detailed review of the genotype and phenotype of all cases with truncating variants of known pathogenicity in DSP. Cardiac magnetic resonance short-axis images (T2-STIR sequence for myocardial oedema, top; late gadolinium enhancement, bottom) are presented for the 3 cases from London and histopathology for the 1 case from Maastricht. Images are taken at 100 µm and demonstrate staining for (A) hematoxylin-eosin, (B) CD45, (C) CD68, and (D) Sirius red. ACM indicates arrhythmogenic cardiomyopathy; FHx, family history; gnomAD, Genome Aggregation Database; Het, heterogeneous; LVEF, left ventricular ejection fraction; P, patient; SCD, sudden cardiac death; and Trop, troponin.