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. 2022 Oct 13;22:318. doi: 10.1186/s12935-022-02634-7

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Expression profile of ferroptosis-related DEGs in normal liver tissue vs. HCC and HCV- induced cirrhosis vs. HCC from GEO datasets. A, B The venn diagrams of the expression of differential gene STEAP3 in normal liver tissue, cirrhosis and HCC tissues. C, D. The volcano diagrams of the expression of differential gene STEAP3 in normal liver tissue, cirrhosis and HCC tissues. E The RT-PCR results showing the expression level of STEAP3 in normal liver tissues and HCC tissues. F The RT-PCR results showing the expression level of STEAP3 in HCV-induced cirrhosis and HCC tissues