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. 2022 Apr 6;29(10):1439–1451. doi: 10.1038/s41417-022-00453-6

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — A The characteristics of healthy negative control serum exosomes and NPC serum exosomes were identified by transmission electron microscopy and Western blotting. B The content of HMGA2 in healthy negative control serum exosomes and NPC serum exosomes with and without metastasis. Densitometry was performed with ImageJ. ***p < 0.001. C The permeability of HUVEC monolayers treated with circulating exosomes from healthy negative controls and circulating exosomes from NPC patients with and without metastasis was measured by the absorption value of rhodamine-dextran. ***p < 0.001. D Representative images (left panel) and histogram of the quantification (right panel) of the endothelial cellular morphology alteration after the cells were treated with serum exosomes from healthy negative controls and serum exosomes from NPC patients with and without metastasis for 48 h. ***p < 0.001.