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. 2021 Oct 13;3:756704. doi: 10.3389/frph.2021.756704

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Copyright © 2021 Kuan, Gibson, Whitaker and Horne.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Summary of dysregulated menstrual pathways that can contribute to the pathogenesis of endometriosis. In women with endometriosis, dysregulation of several menstrual processes (angiogenesis, EMT, apoptotic regulators, inflammation, debris clearance, MMP) may promote endometrial cell detachment, prolong endometrial cell survival, and increase the likelihood for implantation following retrograde menstruation. Understanding these pathways may introduce new therapeutic targets and can be achieved by further exploring the underlying mechanisms of menstrual dysregulation in endometriosis. Created with BioRender.com.