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Macrophage autophagy alleviates liver fibrosis. LC-3-associated phagocytosis could remove apoptotic cells and pathogens to reduce inflammation. With the assistance of LC-3-associated phagocytosis, autophagy could reduce inflammatory cytokines being secreted. With fewer inflammatory cytokines, myofibroblast would produce less collagen and relieve lung fibrosis. ITAMi, inhibitory immunoreceptor tyrosine-based activation motif; LAP, LC3-associated phagocytosis.
