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. 2022 Oct 19;23:437. doi: 10.1186/s12859-022-04984-5

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — The association between m6A-related genes and methylation and the role of m6A-related genes in cancer-related pathways. A Methylation difference between tumor and normal samples in LUAD and LUSC. B Spearman Correlation Coefficient of methylation and gene expression in LUAD and LUSC. C Overall survival difference between hypermethylation and hypomethylation in LUSC. D Global percentage of activity of m6A-related gene pathway in LUAD and LUSC. A represents Activate, while I represents Inhibit. E Interaction map of m6A-related genes and pathway in LUAD and LUSC. Solid line represents activation, while dotted line means inhibition. F Pie percentage of activity of m6A-related gene pathway in LUAD and LUSC. Red part means activation, while green part represents inhibition