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. 2022 Oct 21;41:309. doi: 10.1186/s13046-022-02479-y

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — MET-CAR-T therapeutic activity in experimental tumors overexpressing the MET receptor. (A) Caki-1 tumor growth in NOD-SCID mice treated with 10⁷ MET-CAR-T by intravenous injection on days 4, 7, and 11. (B) GTR-210 tumor growth in NOD-SCID mice treated with 10⁷ MET-CAR-T by intravenous injection on day 6. Vehicle: PBS; NTD: Not-transduced T cells; #948/#949: MET-CAR-T. Bars: Standard Error of the Mean. Statistical significance between Vehicle and MET-CAR-T was calculated by Two-way Anova, corrected Bonferroni. Stars indicated P values: ****, P ≤ 0.0001