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. 2022 Oct 18;13(1):1849–1867. doi: 10.1080/21505594.2022.2136433

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© 2022 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 7. — AP regulates the intracellular and supernatant viral titres of NDVs with a mutated YLMY motif. The viral infectivity was measured as the tissue culture infective dose by inoculating naive cells with lysates (intracellular virus) or supernatants (extracellular virus) from NDV-infected cells in which AP1M1 or AP2M1 had been depleted via siRNA treatment. (a–d) the intracellular (a and b) or extracellular (c and d) virus titer of siAP1M1-treated (a and c) or siAP2M1-treated (b and d) cells infected with an NDV containing a wildtype (rSG10*) or mutated (rSG10*-F/Y524A, rSG10*-F/Y527A, or rSG10*-F/Y524AY527A) YLMY motif.