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. 2022 Apr 27;71(12):2913–2928. doi: 10.1007/s00262-022-03202-8

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Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visithttp://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — Patient #1 a Timeline demonstrating initial diagnosis, status of remission and therapeutic interventions. b Leukocytes, thrombocytes and hemoglobin values during clinical course. c Frequencies of blasts, NPM1 and WT1 normalized copy numbers (NCN) in bone marrow during clinical course. Peripheral blood was used for the measurement of NPM1 NCN at day 384, 468, 825, 923, 1236, 1432, 1614, 1796 and 1985. d Gene mutations at initial diagnosis and relapse of AML measured by next-generation-sequencing (54-myeloid-gene panel)