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. 2022 Oct 11;10:1023006. doi: 10.3389/fcell.2022.1023006

TABLE 1.

Characteristics of selected GWASs.

Trials Cases (N) Controls (N) Sample size (N) Year of publication No. of SNPs
Lipoprotein metabolism a 14,010 197,259 211,269 2021 16,380,413
Coronary artery disease (van der Harst and Verweij, 2018) 122,733 424,528 547,261 2017 7,934,254
Aortic aneurysm a 2,825 206,541 209,366 2021 16,380,417
Heart failure (Shah et al., 2020) 47,309 930,014 977,323 2020 7,773,021
Atrial fibrillation (Nielsen et al., 2018) 60,620 970,216 1,030,836 2018 33,519,037
Hypertension b 54,358 408,652 463,010 2018 9,851,867
Old myocardial infarction b 3,340 459,670 463,010 2018 9,851,867
Acute myocardial infarction b 2,321 460,689 463,010 2018 9,851,867
Stroke b 7,055 454,825 461,880 2018 9,851,867
Peripheral vascular disease b 1,456 461,554 463,010 2018 9,851,867
Venous thromboembolism b 4,620 356,574 361,194 2018 11,901,177
a

Output from the Finn GWAS.

b

Output from GWAS, pipeline using Phesant derived variables from UKBiobank.

GWAS, genome-wide association studies.