TABLE 1.
Characteristics of selected GWASs.
Trials | Cases (N) | Controls (N) | Sample size (N) | Year of publication | No. of SNPs |
---|---|---|---|---|---|
Lipoprotein metabolism a | 14,010 | 197,259 | 211,269 | 2021 | 16,380,413 |
Coronary artery disease (van der Harst and Verweij, 2018) | 122,733 | 424,528 | 547,261 | 2017 | 7,934,254 |
Aortic aneurysm a | 2,825 | 206,541 | 209,366 | 2021 | 16,380,417 |
Heart failure (Shah et al., 2020) | 47,309 | 930,014 | 977,323 | 2020 | 7,773,021 |
Atrial fibrillation (Nielsen et al., 2018) | 60,620 | 970,216 | 1,030,836 | 2018 | 33,519,037 |
Hypertension b | 54,358 | 408,652 | 463,010 | 2018 | 9,851,867 |
Old myocardial infarction b | 3,340 | 459,670 | 463,010 | 2018 | 9,851,867 |
Acute myocardial infarction b | 2,321 | 460,689 | 463,010 | 2018 | 9,851,867 |
Stroke b | 7,055 | 454,825 | 461,880 | 2018 | 9,851,867 |
Peripheral vascular disease b | 1,456 | 461,554 | 463,010 | 2018 | 9,851,867 |
Venous thromboembolism b | 4,620 | 356,574 | 361,194 | 2018 | 11,901,177 |
Output from the Finn GWAS.
Output from GWAS, pipeline using Phesant derived variables from UKBiobank.
GWAS, genome-wide association studies.