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. 2022 Jun 22;28(4):827–840. doi: 10.3350/cmh.2022.0068

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Copyright © 2022 by The Korean Association for the Study of the Liver

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 5. — Auranofin reduced lipogenesis by generating inflammation reduction and antioxidants in PA-induced HepG2 cells. Quantitative comparison of mRNA levels of (A) inflammation, (B) lipogenesis markers, and (C) antioxidant in auranofin treatment results in PA-induced HepG2 cells. (D) Comparison of fat accumulation by auranofin treatment in PA-induced HepG2 cells with Nile-red O staining (×630). Data shown in bar diagrams as mean±standard error of mean from three independent experiments. TNF, tumor necrosis factor; GAPDH, glyceraldehyde-3-phosphate dehydrogenase; PA, palmitic acid; IL, interLeukin; MCP, monocyte chemoattractant protein. ^*P<0.05. ^**P<0.01. ^***P<0.001. ^****P<0.0001.