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. 2022 Oct 15;10(10):2592. doi: 10.3390/biomedicines10102592

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Connections of anti-tuberculosis drugs resistance and protein phosphorylation in MTB. (A) PknB mediates drug resistance/tolerance by phosphorylation; (B) Regulation of STPK genes after treatment with anti-tuberculosis drugs. EF-Tu, elongation factor Tu; EMB, ethambutol; ETO, ethionamide; GTP, guanosine-5-triphosphate; INH, isoniazid; InhA, 2-trans-enoyl-acyl carrier protein reductase; MabA, beta-ketoacyl-acyl carrier protein reductase; MurA, UDP-N-acetylglucosamine enolpyruvyle transferase; NAD+, nicotinamide adenine dinucleotide; P, phosphorylation; RIF, rifampicin; STPK, Ser/Thr protein kinase; SahH, S-adenosyl homocysteine hydrolase.