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. 2022 Oct 26;44(2):437–446. doi: 10.1007/s10072-022-06429-6

Table 1.

Clinical features of COVID-GBS patients

Data (n = 38) Value
Age at onset 60.7 years
Male 89.4%
COVID-19 swab test positivity 33/38
Time interval COVID-19 and GBS 15.1 days
Symptoms
  Lower limbs paresis 73.7%
  Involvement of upper limbs 55.3%
  Tetraparesis 31.6%
  Facial nerve involvement 55.3%
  Sensory loss and paresthesia 36.8
  Hypo-areflexia 100%
  Miller-Fisher syndrome 5.26%
COVID-19
  Severe 68.4%
  Mild/moderate 31.6%
CSF (n = 18)
  Albuminocytological dissociation 71.4%
  SARS-COV-2 negative 15/15
Electrophysiological findings 33/38
  Reduced nerve conduction velocity (NCV) 65.7%
  Altered/absent F-wave 82.8%
  Increased distal latency 60%
  Conduction blocks 5.2%
Diagnosis
  Acute inflammatory demyelinating polyneuropathy (AIDP) 81.8%
  Acute motor and sensory axonal neuropathy (AMSAN) 12.1%
  Acute motor axonal neuropathy (AMAN) 6.0%
MRI lumbosacral spinal cord with gadolinium
  Increase in size of lumbosacral roots 2/14
Treatment
  Intravenous immunoglobulin (IVIg) 29/38
  Plasma exchange (PE) 2/38
  IVIg and plasma exchange 2/38
  Untreated 5/38
Clinical course at follow-up
  Favorable 76.3%
  Stable 10.5%
  Worsening 13.2%
  Death 8.1%
Respiratory involvement
  Orotracheal intubation and invasive ventilation 52.6%
COVID-19 consequences
  No detectable pulmonary complications 26.3%
  Minor pulmonary complications 21%
  Severe with a bilateral pulmonary involvement with respiratory distress 52.6%
Used therapy
  Hydroxychloroquine 63.1%
  Tocilizumab 15.8%
  Lopinavir/ritonavir 18.4%
  Azathioprine 5.3%
  Steroids 44.7%
  Prophylactic anticoagulant with LMWH 84.2%

GBS, Guillan-Barrè syndrome; LMWH, low-molecular weight heparin