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. 2022 Oct 28;101(43):e31416. doi: 10.1097/MD.0000000000031416

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Copyright © 2022 the Author(s). Published by Wolters Kluwer Health, Inc.

This is an open-access article distributed under the terms of the Creative Commons Attribution-Non Commercial License 4.0 (CCBY-NC), where it is permissible to download, share, remix, transform, and buildup the work provided it is properly cited. The work cannot be used commercially without permission from the journal.

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Figure 5. — The landscape of mutations among PCa metabolic subgroups.This heat map showed the distribution of somatic mutation (SNP) (A) and CNV (B) events that affected frequently mutated genes across metabolic subgroups in PCa. (C) Mutation analysis between the glycolytic and cholesterogenic subsets. (D) Gene mutation map of highly mutated genes between the 2 subtypes. (E) The forest plot exhibited a comparison of gene mutations between the 2 subtypes (*P: .1, **P: .05, ns: not significant). PCa = prostate cancer.