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. 2022 Nov 1;13(11):919. doi: 10.1038/s41419-022-05362-y

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a m6A level in patients with or without OP (both n = 30). b The levels of BGLAP, COL1A1, RUNX2, OCN, and OPN were detected using qRT-PCR. c The protein levels of BGLAP, COL1A1, RUNX2, OCN, and OPN in the differentiated and undifferentiated BMSCs. d m6A level in osteoblast differentiated or undifferentiated cells. e The mRNA levels of BGLAP, COL1A1, RUNX2, OCN, and OPN were detected in mice of the sham and OVX groups. f The protein levels of BGLAP, COL1A1, RUNX2, OCN, and OPN in the sham and OVX mice. g Images of trabecular bone of mice. h Bone formation in mice was measured using the H&E assay and i BS was quantified. Bone formation index containing j BV/TV, k BMD, l Tb.N, m Tb.th, and n Tb.Sp were analyzed by μ-CT. o m6A levels in mice were tested using qRT-PCR. **P < 0.01 and *P < 0.05.