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. 2022 Nov 1;13(11):919. doi: 10.1038/s41419-022-05362-y

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a The mRNA levels of Notch1, Jagged1, HEY1, PI3K, AKT, mTOR, SMAD1, Smad5, Smad8, Wnt1, β-catenin, and GSK3β after transfection with METTL14. b m6A modification of Smad1, Smad5, and Smad8 following sh-METTL14 transfection. c SMAD1 expression in osteogenic differentiated and undifferentiated BSMCs. d SMAD1 expression in sham and OVX mice. e SMAD1 expression in patients with or without OP (both n = 30). f The relationship between METTL14 and SMAD1 was assessed by Pearson correlation coefficient (g) SMAD1 related proteins were predicted using the STRING online tool. **P < 0.01.