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. 2022 Sep 1;26(5):434–439. doi: 10.4103/jisp.jisp_67_21

Table 2.

Differential diagnosis of Plasma cell gingivitis

Clinical differentials
Histological differentials
Cicatricial pemphigoid (CP) discoid lupus erythematosus Pyogenic granuloma Oral lichen planus Plasma cell dyscrasias
IgG4^ related disease
Extramedullary plasmacytoma Plasma cell Myeloma with extramedullary involvement
Clinical entity presenting granulomatous overgrowth Mucocutaneous lesions, vesicles, Nikolsky's sign Pedunculated lesion involving gingiva most frequently Bilaterally symmetrical lesion involving skin, buccal mucosa most frequently Head and Neck most common, Soft tissue mass Local growth of soft tissue in vicinity to skeleton or hematogenous spread to any other organ or tissue Heterogeneous clinical and radiological presentation which mimics malignancy.
Histology Subepithelial split with variable inflammation in CP*
Hyperkeratosis, follicular plugging, degeneration of basal cells
Abundant engorged blood vessels in the background of mixed inflammatory infiltrate Saw toothed rete ridges, liquefactive degeneration of basal layer with intense lymphocytic infiltrate subepithelially monoclonal plasmacytoid cells with light chain restriction Similar to plasmacytoma, acquire blastoid morphology in plasmablastic plasma cell myeloma Storiform fibrosis, obliterative phlebitis and more than10/high power field IgG4 positive plasma cells (more specifically ratio of IgG4 to IgG positive plasma cells as >0.4)
Treatment Systemic corticosteroid, immunomodulator therapy Conservative surgical excision Symptomatic cases require variable treatment ranging from topic corticosteroids to antifungal or systemic steroids or immunomodulators Radiation therapy Chemotherapy Corticosteroids
Prognosis Generally good, spontaneous remission in 2-5 years Excellent, occasional recurrence Malignant potential controversial Better prognosis than solitary plasmacytoma, 30% patient progress to PCM# Usually incurable progressive disease, median survival rate 5.5 years Irreversible injury to some organs if therapy is not initiated in time, once fibrosis is established treatment options are limited

CP - Cicatricial pemphigoid, PCM - Plasma cell myeloma, IgG - Immunoglobulin G