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. 2022 Oct 19;13:1015221. doi: 10.3389/fneur.2022.1015221

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Copyright © 2022 Wang, Wei, Long, Wang, Yang, Wu, Li, Li, Arshad, Li and Chen.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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GSEA, GO analysis, and KEGG enrichment analysis reveal the potential mechanism by which CENPA promotes glioma progression using the CGGA database (LGG+GBM). The top 8 gene ontologies (A) and KEGG pathways (B) based on the normalized enrichment score (NES) between the high- and low-CENPA expression phenotypes. (C) Pearson correlation coefficient heatmap of the top 100 genes positively related to CENPA. The top 10 KEGG pathways (D) and biological processes, cellular components, and molecular functions (E) of the top 100 genes co-expressed with CENPA in gliomas.