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. 2022 Nov 1;15:229. doi: 10.1186/s12920-022-01387-6

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Graphs and diagram related to MS-HRM analysis of DIO3 gene promoter methylation. Aligned melt curves for DIO3 controls (a) and normal-weight compared to obese samples (b). There was a significant difference in mean ± SD methylation levels at DIO3 promoter region between obese and normal-weight samples. c *P < 0.001 between normal-weight and obese subjects